As expected, TEPP-46 was capable of enhancing PKM2 and GLUT1 expression in Sal B-exposed NCI-H2030 as well as NCI-H1650 cells (Figures 7(a)–7(j)), demonstrating that TEPP-46 markedly reversed the inhibitory effects of Sal B on metabolic reprogramming of NSCLC cells. The gene discussed is PKM; the disease is non-small cell lung carcinoma.