Drusen have also been described in many other forms of glomerulonephritis (GN), namely, dense deposit disease, membranous and poststreptococcal GN,11,28 and atypical haemolytic uremic syndrome,29,30 which are all associated with risk alleles in CFH or other complement pathway genes (CFH, CFI, C3) and with complement activation.31 The gene discussed is CFH; the disease is glomerulonephritis.