Previous studies have demonstrated that CPEB1 acts as a major regulator of stress granule (mRNA-ribonucleoprotein complex) formation, and modulates translational efficiency under stress conditions, including heat shock (Namkoong et al., 2018), hypoxia (Arimoto et al., 2008), and viral infection (McCormick and Khaperskyy, 2017). Here, CPEB1 is linked to viral infectious disease.