Transcriptional analysis of the Kasumi-1 AML cell line as early as 14 h post-transduction with a vector coding for NR4A1 or NR4A3 revealed the redundancy of these proteins in this specific context, with 97% of genes commonly regulated (Boudreaux et al., 2012). This evidence concerns the gene NR4A3 and acute myeloid leukemia.