These results are congruent with a clinical study showing that plasma IL-17A levels correlated with ED in RA [44], and with a recent study in a cohort of 36 RA patients with a follow-up between 1970 and 2012 indicating that high levels of IL-17A were associated with both joint destruction and the occurrence of myocardial infarction [45]. This evidence concerns the gene IL17A and myocardial infarction.