Mutations in tyrosinase (TYR), oculocutaneous albinism type 2 (OCA2), melanocortin 1 receptor (MC1R), tyrosinase-related protein 1 (TYRP1), solute carrier family 45 member 2 (SLC45A2), solute carrier family 24 member 5 (SLC24A5), and chromosome 10 open reading frame 11 (C10ORF11) can cause non-syndromic OCA, which can be broadly classified into several subtypes—OCA type 1, OCA type 2, OCA type 3, OCA type 4, OCA type 6, and OCA type 7 [1, 4]. This evidence concerns the gene LRMDA and oculocutaneous albinism.