FOXP3 and glioblastoma: Interestingly, for Cluster 7 macrophages that derived by TLR4 deficiency, a Foxp3‐centric “Foxp3/CEBPb/Nur77(Nr4a1)” network turned out to be the mostly enriched signalling pathway (Figure 4E), indicating the immune regulatory role of this unique macrophage subtype in anti‐GBM cGN.