Interestingly, for Cluster 7 macrophages that derived by TLR4 deficiency, a Foxp3‐centric “Foxp3/CEBPb/Nur77(Nr4a1)” network turned out to be the mostly enriched signalling pathway (Figure 4E), indicating the immune regulatory role of this unique macrophage subtype in anti‐GBM cGN. Here, CEBPB is linked to glioblastoma.