FGFR4 and glioblastoma: Unsupervised GSEA of both tested GBM models, BTL1528 (Fig. 5A–C, Additional File 1: Figure S10A, Additional File 7: Table S5) and BTL1376 (Additional File 1: Figure S9A–C, Additional File 8: Table S6), further revealed alterations in REACTOME pathways including extracellular matrix organization, NCAM signaling for neurite outgrowth, and collagen formation following FGFR4 inactivation.