In addition, genes associated with hematological disease (e.g., PDE7A, LMAN1, F13A1, OLR1) and mitochondrial biogenesis and redox (e.g., NOS3, ALDH5A1, PRXL2A, SLC25A13, CPT2) were also significantly altered in BPD patients (Table 4). This evidence concerns the gene PRXL2A and bronchopulmonary dysplasia.