REN and infarction: When MI occurs, cardiac injury signals are “sent” to stellate ganglia and result in increased cardiac sympathetic innervation and activation of the renin-angiotensin aldosterone system, which plays an essential role in the genesis and maintenance of ventricular arrhythmias, not only in the acute phase of myocardial ischaemia but also in the post-infarction remodelling process [38, 39].