While future research is required to determine whether elevated levels of phenolics that stimulate and inhibit Fe uptake in the human gut can help alleviate the anaemia seen in Ethiopian children [67], the significance of the SNP in the tef F3’H gene Et_s3159-0.29-1.mrna1, which explains a large proportion of the variation in kaempferol glycoside and quercetin glycoside levels, suggest this might be a breeding target to improve Fe bioavailability in tef, as kaempferol glycoside is known to promote Fe absorption while quercetin glycoside inhibits Fe absorption in cell assays [60]. This evidence concerns the gene TEF and anemia (phenotype).