STING1 and autoimmune interstitial lung disease-arthritis syndrome: Considering that loss of COPA is associated with Golgi dispersal41 (Supplementary Fig. 4, 5) and spontaneously accumulated STING-GFP co-localized with Golgi but not ER markers, these experiments suggest that STING signalling occurs due to spontaneous accumulation at the dispersed Golgi in this in vitro model of COPA syndrome.