Compound heterozygosity for α0- and α+-thalassemias leads to the Hb H disease, and interaction of Hb H disease with Hb E can lead to the Hb AEBart’s and EFBart’s diseases, the thalassemia intermedia commonly encountered in the region [8, 9]. This evidence concerns the gene GSTM1 and beta-thalassemia intermedia.