Furthermore, Wnt5a expression was significantly downregulated in FAP-isolated from mdx mice, an animal model of Duchenne muscular dystrophy, which is prone to adipogenesis in dystrophic muscles, and exogenous administration of Wnt5a prevented the in vitro adipogenesis of isolated FAPs from mdx mice, thus confirming that Wnt5a inhibits adipogenesis. The gene discussed is WNT5A; the disease is Duchenne muscular dystrophy.