There are several interpretations on Wnt-mediated inhibition of GSK3β in different models, including dissociation of GSK3β from AXIN via conformational changes [55] or posttranslational modifications [56], recruitment of GSK3 inhibitory proteins such as frequently rearranged in advanced T-cell lymphomas (FRATs) [57], or degradation of AXIN [58]. The gene discussed is GSK3B; the disease is T-cell non-Hodgkin lymphoma.