They also added that monovalent EGFR-redirected targeting modules could only induce antitumor activity when they encountered high-level EGFR expression on tumor cells while bivalent α-EGFR-EGFR-redirected targeting modules could meditate UniCAR-T-induced antitumor activity towards cancer cells expressing low levels of EGFR [109]. Here, EGFR is linked to neoplasm.