An excessive inflammatory response to SARS-CoV-2 is a major cause of disease severity as well as mortality in COVID-19 patients and is associated with high levels of circulating cytokines i.e., IL-1β, IL-6, IL-7, IFNγ and TNFα and chemokines i.e., CCL2, CLL3 and CXCL10 (1, 41, 42). This evidence concerns the gene CCL2 and COVID-19.