PINK1 and diabetic cardiomyopathy: AMPKα2 interacted with PTEN-induced putative kinase 1 (PINK1) and enhanced the activity of PINK1-Parkin-sequestosome-1 pathway involved in cardiac mitophagy, which led to elimination of damaged mitochondria, improvement in mitochondrial function, decrease in reactive oxygen species production, and apoptosis of cardiomyocytes.163 miR-342-5p targeted Caspase 9 and JNK2 to inhibit I/R-induced cardiomyocyte apoptosis.164 miR-320 promoted cardiomyocyte apoptosis and exacerbated cardiac function in a model of diabetic cardiomyopathy.