These chemokines have been implicated in the induction and perpetuation of several viral infections by mediating CD4+ T-cell recruitment, activation, and function.15–17 Given that recruitment of CD4+ T cells to infected areas may be one of the mechanisms that account for virus-specific CD4+ maintenance, we investigated the expression of SAR-CoV-2 infection-induced CXCR3 ligands CXCL9, CXCL10, and CXCL11. This evidence concerns the gene CXCL9 and viral infectious disease.