Among multiple prostate cancer cell lines, spanning diverse molecular subtypes both in terms of AR expression as well as position along the cEMT spectrum, we found that IFNγ upregulated MHC-I expression both in vitro and in vivo, and PD-L1 expression was upregulated in vitro (corroboration in vivo was not technically possible). The gene discussed is AR; the disease is Familial prostate cancer.