Exogenous palmitate promoted cell-cycle arrest, DNA damage, autophagy, and apoptosis in two human endometrial epithelial cell lines [26]; suppressed cell-membrane fluidity and glucose metabolism in hepatocellular carcinoma cells [27]; induced cell-cycle G2/M arrest and promoted apoptosis in human neuroblastoma cells and breast cancer cells [28,29]; induced a different transcription program in breast cancer, reducing expression of HER2 and HER3 [30]; and showed selective toxicity and induced apoptosis in leukemia cell lines [31]. This evidence concerns the gene ERBB2 and breast carcinoma.