IL6 and COVID-19: It is accompanied by the release of many proinflammatory cytokines such as TNF-α, interleukin (IL)-6, and IL-1β, which are related to vascular hyperpermeability, lung injury, multiorgan failure, and COVID-19 severity, followed by hyper-reaction and cytokine storm in some patients, which can develop into a pathogenic process of acute respiratory disease syndrome [12].