Moreover, the more abundant expressions of the receptors of those cytokines (Il-17 receptors a–e, IL-22 receptors a1 and a2, IFNg receptors Ifngr1 and Ifngr2), combined with other DEGs, helped to drive the enrichment categories “IL-17 signaling pathway,” “Th17 cell differentiation,” and “response to interferon-gamma,” which can indirectly serve as a proof of the principle that cytokines were induced during peak infection. The gene discussed is IL17A; the disease is infection.