The discovery of the causative or susceptibility genes of various fPD, since the end of 20th century, has greatly promoted the elucidation of molecular mechanism of sPD [3]; fPD is termed in the order of discovery of the gene loci such as PARK1 (α-synuclein, SNCA [9,10]) and PARK2 (parkin, PRKN [3,11,12]). The gene discussed is PRKN; the disease is Platelet storage pool disease.