Mutations in some genes in fPD are considered to be causative and also related to susceptibility loci in sPD, for example, α-synuclein gene (SNCA and PARK1) [9,10], parkin (PARK2) [3,11], PTEN-induced putative kinase 1 (PINK1 and PARK6) [13,14], and leucine-rich repeat kinase 2 (LRRK2 and PARK8) [15,16,17,18]. Here, PRKN is linked to Platelet storage pool disease.