FKBP11 and idiopathic pulmonary fibrosis: Our correlations of FKBP11 transcript abundance with lung function and transcript abundance of typical fibrotic markers in IPF lungs suggest that there is no strong direct relationship between the level of scarring and the presence of FKBP11+ plasma cells (Figure S4), but a time- and space-resolved analysis of interstitial scarring and lung-resident plasma cell differentiation would be necessary to draw robust conclusions in terms of cause or consequence.