The conclusions were as follows: (1) 6 weeks of exercise intervention during the recovery phase of ALD may ameliorate hepatocyte damage and dyslipidemia through the IL-6–p47phox oxidative–stress axis, and administering apocynin in combination could optimize this effect; (2) Drinking alcohol during exercise exacerbates dyslipidemia, oxidative stress, and downregulated expression of the IL-6–p47phox axis–related protein in hepatocytes. Here, NCF1 is linked to metabolic syndrome.