In addition, we showed that the facultative glucose transporter-3 (GLUT3) was overexpressed in PE [26]; if LRRC27/42 functioned similarly as LRRC8A, both may operate in tandem with GLUT3 to enhance ‘resting state’ glucose entry in platelets and contribute to the prothrombotic tendency of preeclampsia. Here, SLC2A3 is linked to preeclampsia.