LRRC8A and preeclampsia: In addition, we showed that the facultative glucose transporter-3 (GLUT3) was overexpressed in PE [26]; if LRRC27/42 functioned similarly as LRRC8A, both may operate in tandem with GLUT3 to enhance ‘resting state’ glucose entry in platelets and contribute to the prothrombotic tendency of preeclampsia.