Previous studies revealed that the concentration of the chemokines MIP-1α/CCL3 [22], eotaxin/CCL11 [23], TARC/CCL17 [24], and PARC/CCL18 [25] in tissue samples obtained from CRSwNP patients were higher than in patients with CRSsNP. The gene discussed is CCL3; the disease is chronic rhinosinusitis without nasal polyps.