Specifically for MSA, upregulation of miR-96 with gene targets, including FOXO, SOX, FYN, neuregulin, and SLC proteins (SLC1A1 and SLC6A6), could be involved in neurodegeneration, such as oxidative damage, zinc dyshomeostasis, and excitotoxicity [23]. Here, SLC1A1 is linked to multiple system atrophy.