The Cx37 C1019T polymorphism has been linked to a variety of human diseases including coronary heart disease (CHD), peripheral artery disease, in-stent restenosis, myocardial infarction (MI) and stroke [93,94,95,96,97], most likely through regulating the adhesion of monocytes [98]. Here, GJA4 is linked to myocardial infarction.