Although the exact molecular mechanisms were not clarified, the authors suggested that Hsp27-mediated upregulation of Snail resulted in cytoskeletal changes such as a decrease in E-cadherin, eventually promoting the EMT of pancreatic cancer cells, while the increased expression of ERCC1 has been associated with nucleotide excision repair after chemotherapy [51,98,105]. The gene discussed is HSPB1; the disease is pancreatic neoplasm.