CXCR5-CXCL13 interaction has been shown to play a role in the maintenance of pathogenic B cells in several autoimmune diseases, including rheumatoid arthritis, Sjögren’s syndrome, Hashimoto thyroiditis, Graves’ disease, multiple sclerosis, and myasthenia gravis [28]. The gene discussed is CXCR5; the disease is Hashimoto thyroiditis.