Genes that were found to be upregulated in the early COVID-19 mortality cases and involved with the interferon (ISG20, IRF1, GBP2) and NF-κB (NFKB2, NFKBIA, TNFAIP3) pathways showed linear decline with longer symptomatic interval (Figure 2C). Here, NFKB1 is linked to COVID-19.