As shown in Figure 10, compared with the S180 tumor-bearing mice model group, the expression of the antiapoptotic factor Bcl-2 was decreased in the AGS-H and AGS-H synergistic with CTX/2 groups (P < 0.05, P < 0.001), which significantly increased the expression of the apoptosis factors Bax and cleaved-Caspase-3 (P < 0.05, P < 0.001) in a dose-dependent manner. Here, BCL2 is linked to neoplasm.