They could decrease the expression and phosphorylation of epidermal growth factor receptor (EGFR), Met, Akt, and NF-κB in cervical cancer cells, and totally inhibit the EGFR/Met-Akt/NF-κB signaling pathway activated by hepatocyte growth factor (HGF) and epidermal growth factor (EGF) (69). The gene discussed is MET; the disease is cervical cancer.