The oAd/DCN/LRP co-expresses decorin (DCN), which is a leucine-rich proteoglycan that suppresses the production of ECM components by inhibiting the activity of TGF-β, and soluble Wnt decoy receptor (LRP), which prevents the Wnt/β-catenin signaling pathway from promoting epithelial-to-mesenchymal transition (EMT) and metastasis of the tumor [57–59]. This evidence concerns the gene DCN and neoplasm.