In the high-inflammatory response group, we observed that pro-inflammatory genes such as CCL2, CCL3, CCL4, TNF-α, IFN-γ, andIL-6are generally highly expressed, and many studies have confirmed that they promote the survival and reproduction of AML cells [7, 26–28]; we also noticed that classical anti-inflammatory cytokines such as IL-10 and TGF-β showed high expression levels, which tend to inhibit the proliferation of AML cells, but these studies were for exogenous cytokines such as in plasma. This evidence concerns the gene CCL2 and acute myeloid leukemia.