TGFB1 and acute myeloid leukemia: In the high-inflammatory response group, we observed that pro-inflammatory genes such as CCL2, CCL3, CCL4, TNF-α, IFN-γ, andIL-6are generally highly expressed, and many studies have confirmed that they promote the survival and reproduction of AML cells [7, 26–28]; we also noticed that classical anti-inflammatory cytokines such as IL-10 and TGF-β showed high expression levels, which tend to inhibit the proliferation of AML cells, but these studies were for exogenous cytokines such as in plasma.