A few studies identified higher CRP levels in patients with VTE (mainly DVT), [42] whereas the study by Antic et al. [43] published results similar to ours, showing the effect of a broad inflammatory and hemostatic biomarker spectrum (including D-dimer, Factor XIIIa, von Willebrand factor, TNF-α, protein S, β2Glycoprotein I, MPs, urokinase-like plasminogen activator, fibronectin, and plasminogen activator inhibitor type 1). The gene discussed is VWF; the disease is deep vein thrombosis.