ADAMTS2 and osteogenesis imperfecta: These defects are likely to be early onset and to impair alveolar formation, similar to what was described in Adamts2‐/‐ mice (a mouse model of dermatosparaxis dEDS) (Le Goff et al., 2006) and in mouse models of OI with alterations in type I collagen (Baglole et al., 2018; Baldridge et al., 2010; Dimori et al., 2020; Grafe et al., 2014).