Anterior segment photography (Figure 4(a)) and clinical scores (galectin-3−/− FK vs. wild type FK D1: 5.60 ± 0.15 vs. 5.50 ± 0.10; D3: 8.70 ± 0.20 vs. 10.60 ± 0.25; D5: 11.50 ± 0.20 vs. 11.90 ± 0.10) (Figure 4(b)) showed that compared with the wild type FK group, the condition of galectin-3−/− FK group aggravated rapidly on the third day after infection, mainly caused by increased perforation rate (galectin-3−/− FK vs. wild type FK D1: 1.33 ± 0.58 vs. 1.67 ± 0.58; D3: 2.33 ± 0.58 vs. 3.67 ± 0.58; D5: 3.33 ± 0.58 vs. 4.00 ± 0.00) (Figure 4(e)). Here, LGALS3 is linked to infection.