Moreover, Pedersen et al. [80] demonstrated that miR-155 targeted SHIP1 to promote TNF-α-dependent B cell lymphomas growth, and upregulated miR-155 and consequently downregulated SHIP1 expression brought about autocrine stimulation by the proinflammatory cytokine TNF-α in DLBCL. Here, TNF is linked to B-cell non-Hodgkin lymphoma.