The functional enrichment of the DEGs indicated that miR-29c might be a potential regulator of oxidative stress and inflammatory response in bMECs through multiple genes, such as FOXO1, TNF-α, and BoLA-DQA5, which enriched in stress-activated MAPK cascade, Epstein-Barr virus infection and inflammatory bowel disease signaling pathways. This evidence concerns the gene TNF and Epstein-Barr virus infection.