Takai et al. reported similar therapeutic effects and detailed mechanisms of NF-κB signaling pathway inhibitors (parthenolide inhibits NF-κB signaling pathway by suppressing both the phosphorylation of IκB kinase complex and IκBα degradation) for endometriosis mice, including inhibition effect for number, the weight of endometriotic lesions, the level of vascular endothelial growth factor (VEGF), monocyte chemotactic protein-1 (MCP-1), and leukemia inhibitory factor (LIF) gene expression, which established the basis for our experimental comparison (27). Here, CCL2 is linked to endometriosis.