In the vitiligo mouse model, blockade of IFN-γ, IFNGR (receptor of IFN-γ), JAK1/2 (downstream intracellular signal transducer of IFNGR), CXCL10, and CXCR3 suppressed the MSA-specific CD8+ T cell skin infiltration, thereby not only preventing vitiligo progression but also reversing vitiligo, indicating that the IFN-γ-chemokine axis is a potential therapeutic target (22). Here, CXCL10 is linked to vitiligo.