Point mutated p65 at Thr322 or Thr352 in HeLa cells decreased CXCR4 expression compared to transfection with wild-type p65, indicating site specific p65 O-GlcNAcylation contributes to the regulation of CXCR4 expression in cervical cancer cells (82). Here, CXCR4 is linked to cervical carcinoma.