The results from the RT–PCR assay showed that H37Ra infection significantly enhanced the expression of LDLR, ABCG1, and ABCA1 compared to the non-infection control group; the PPARγ agonist BRL49653 significantly increased the expression of CD36, NCEH1, ABCG1 and SR-B1, and the PPARγ antagonist GW9662 downregulated the expression of ABCG1 (Figure 2A). The gene discussed is LDLR; the disease is infection.