Many previous reports showed that expressions of TLR2, TLR3, and TLR4 as proatherogenic receptors were related to the extent and severity of coronary heart disease and led to the development of the atherosclerotic lesions, atherosclerotic arterial occlusions, and acute myocardial infarction [31, 32]. This evidence concerns the gene TLR4 and acute myocardial infarction.