Many previous reports showed that expressions of TLR2, TLR3, and TLR4 as proatherogenic receptors were related to the extent and severity of coronary heart disease and led to the development of the atherosclerotic lesions, atherosclerotic arterial occlusions, and acute myocardial infarction [31, 32]. Here, TLR2 is linked to acute myocardial infarction.