FUNDC1 and myocardial infarction: Therefore, considering the abovementioned researches and the fact that Fundc1 is involved in the response to hypoxia and in autophagy, we established an animal model of HA to explore hypoxic conditions inducing mitophagy and contributing to the removal of damaged mitochondria, thus maintaining myocardial cellular homeostasis and function, and to identify potential targets for improved MI therapies.