A small number of Myf5-Ispd-cKO mice showed incomplete loss of α-DG glycosylation and a much milder muscle phenotype (Supplementary Fig. 2c, d), consistent with previously reported variation in the individual phenotypes of other models of dystroglycanopathy generated using Myf5-Cre KI mice23,24. The gene discussed is MYF5; the disease is neuromuscular disease caused by qualitative or quantitative defects of alpha-dystroglycan.