A recent study suggested MBNL1 could suppress breast cancer metastatic colonization and stabilize metastasis suppressor transcripts (Fish et al. 2016), while another study revealed that MBNL1 was a cancer-related splicing regulator which acted as a splicing repressor in Dicer1 processing, resulting in colorectal carcinogenesis (Tang et al. 2015). This evidence concerns the gene MBNL1 and breast carcinoma.